SMG5, possibly SuperMarioGlitchy5, was a one-time character that appeared once in the SMG4 series. Despite having the same abbreviation, he has nothing to do with the original Super Meme Guardians nor the Super Mega Gangsta Club.

Protein SMG5 is a protein that in humans is encoded by the SMG5 gene. [5] [6] [7] This protein contains a PIN domain that appears to have mutated the residues in the active site. [8]

[Season 5 - Revenge of SMG3 - YouTuber Arc] After the Anime War against Francis, the SMG5 gang can finally rest after saving Desti from a cruel fate and stopping an anime ban caused by the former ruler of the Mushroom Kingdom, Ex-Princess Peach.

Mar 30, 2025 · SMG5 (SMG5 Nonsense Mediated MRNA Decay Factor) is a Protein Coding gene. Diseases associated with SMG5 include Pancreatic Adenosquamous Carcinoma and Carnitine Palmitoyltransferase I Deficiency. Among its related pathways are Peptide chain elongation and Processing of Capped Intron-Containing Pre-mRNA.

Aug 17, 2024 · SMG5 is a key NMD factor. In this study, we generated an Smg5 conditional knockout mouse model and found that Smg5 -null results in early embryonic lethality before E13.5. Furthermore, we produced multiple lines of Smg5 knockout mouse embryonic stem cells (mESCs) and found that the deletion of Smg5 in mESCs does not compromise cell viability.

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SuperMarioGlitchy5 mainly (SuperMemeGuardian5) is a next counterpart of SMG4 and the character made only appearance in SMG4 series. He is also indirectly mentioned by SMG1 as SuperMemeGuardians in end of WOTFI 2021 video. Also SMG5 loves Krab Hot Sauce Super Hot Spicy Fries Melon. The (TOTALLY...

Aug 1, 2024 · We demonstrated an elevated expression of SMG5 in HCC and scrutinized its potential as a therapeutic target. Our findings revealed that SMG5 knockdown not only inhibited the migration, invasion, and proliferation of HCC cells but also influenced sorafenib resistance.

Smg5 encodes a protein involved in mRNA catabolism. [Date last reviewed: 2019-09-19] (FlyBase Gene Snapshot)

SMG5 mutants with a deletion of the conserved C-terminal region or an asp860-to-ala substitution were able to bind UPF1, but they failed to promote UPF1 dephosphorylation. The SMG5 mutants also inhibited nonsense-mediated mRNA decay in a dominant-negative manner.