Feb 18, 2025 · KIM-1 was measured in plasma at baseline (C1D1) and at C3D1 using a Luminex microbead-based assay and an MSD electrochemiluminescence-based assay. KIM-1 values were categorized into high and low according to an optimized cutoff based on the maximum log-rank Q-statistic for PFS.

Oct 5, 2016 · Here we report the de novo assembly and haplotype phasing of the Korean individual AK1 (ref. 1) using single-molecule real-time sequencing 2, next-generation mapping 3, microfluidics-based linked...

May 29, 2024 · These data suggest that circulating KIM-1 may be a non-invasive marker of MRD and disease recurrence and be associated with benefit from atezo in adj RCC. Further investigation of KIM-1 in adj RCC is warranted.

In this review, we focus on describing the mechanisms of renal tubular cells cisplatin-induced apoptosis, the associated inflammatory response and oxidative stress and the role of KIM-1 as a possible biomarker used to predict cisplatin associated AKI. Keywords: kidney injury molecule-1, acute kidney injury, cisplatin nephrotoxicity. 1. Introduction

A red blood cell-derived extracellular vesicle platform targeted Kim-1 in acutely injured mouse kidney and delivered siRNAs for transcription factors <i>P65</i> and <i>Snai1</i>, alleviating inflammation and fibrosis in the tubules.

KIM-1 possesses the properties of ideal marker of renal proximal tubule epithelium injury [70]: in the normal kidney, KIM-1 expression is determined in trace quantities; in ischemic or toxic kidney injury, activation of KIM-1 synthesis in the cells of the damaged tubules and its increased expression on the apical cell membrane is observed ...

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Aug 22, 2023 · Videos you watch may be added to the TV's watch history and influence TV recommendations. To avoid this, cancel and sign in to YouTube on your computer.Kim Kaphwan (OHMSBY Styled) by Joey S.

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May 4, 2021 · Here, we report that KIM-1 mediates PT uptake of palmitic acid (PA)-bound albumin, leading to enhanced tubule injury with DNA damage, PT cell-cycle arrest, interstitial inflammation and fibrosis, and secondary glomerulosclerosis.