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LPS-induced inflammation modulates selective tau phosphorylation. Unlike the minimal effect on APP, we found that the LPS treatment significantly exacerbated the tau pathology in the 3xTg-AD mice.

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Jul 25, 2018 · Using lipopolysaccharide (LPS) preconditioning (PC) as an approach to induce ischemic tolerance in mice, we found generation of neuroprotective monocytes within the spleen, from which they traffic to the brain and meninges, suppressing postischemic inflammation.

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Lipopolysaccharide (LPS) preconditioning provides neuroprotection against subsequent cerebral ischemic injury through activation of its receptor, Toll-like receptor 4 (TLR4). Paradoxically, TLR activation by endogenous ligands after ischemia worsens stroke damage.

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Sera containing higher levels of LPS (only 3 of which are shown in Table 1, numbers 1, 10, and 22) resulted in the appearance of “typical” astrocyte cultures (Fig. la).

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