‪Professor of Pathology and Laboratory Medicine, Emory University‬ - ‪‪Cited by 24,274‬‬ - ‪neuroscience‬ - ‪cancer biology‬ - ‪drug development‬

Dr. Ye is the recipient of numerous professional honors, including the Distinguished Scientist Award from the Sontag Foundation (2003) and American Cancer Research Scholar (2004), and he is also one of the semi-finalists for Keck Foundation (2004).

叶克强教授，深圳理工大学生命健康学院系主任、讲席教授，国家特聘专家，教育部“长江学者”讲座教授，曾任美国埃默里大学终身教授、博士生导师。 深耕神经退行性疾病机制、早期诊断 …

Objective This study is to investigate the role of gut dysbiosis in triggering inflammation in the brain and its contribution to Alzheimer’s disease (AD) pathogenesis. Design We analysed the gut microbiota composition of 3×Tg mice in an age-dependent manner.

Feb 15, 2024 · Dr. Keqiang Ye is currently an endowed professor and Department of Biology Chairman at Shenzhen Institute of Advanced Technology (SIAT), China. Prior to this role, he held positions at Emory University in Atlanta, Georgia, USA, serving as an Assistant Professor (2001–2007), a tenured Associate Professor (2007–2010), and a Full Professor ...

Keqiang Ye is a professor of the Department of Pathology and Laboratory Medicine at Emory. His research interests center upon understanding how nerve growth factor (NGF) regulates neuronal cell survival in the nucleus but also working on drug discovery in the neuroprotection field.

Dr. Ye's lab at Emory University studied neurotrophin signalings in neuronal cell survival and drug discovery in neuroprotection to dissect the physiological roles of PIKE in tumorigenesis and anti-cancer drug development.

In the current paper, researchers led by Keqiang Ye, PhD demonstrated that alpha-synuclein binds and interferes with TrkB, the receptor for BDNF (brain derived neurotrophic factor).

Keqiang YE, Professor (Full) | Cited by 18,401 | of Chinese Academy of Sciences, Beijing (CAS) | Read 367 publications | Contact Keqiang YE

Keqiang Ye: The C/EBPb/AEP pathway is the key driver for Alzheimer's disease (AD) and Parkinson's disease (PD) pathogenesis and its specific inhibitor attenuates AD/PD pathologies