Aug 7, 2023 · AXIN1 knockdown reduced the production of IL-1, IL-6, and TNF in I/R-stimulated AC16 cells, implying that AXIN1 silencing inhibited cell inflammation . Figure 2: Knocking down …

May 1, 2022 · Blocking SOX2OT suppressed oxidative stress and inflammatory response in LPS-treated AC16 cardiomyocytes, accompanied with apoptosis promotion. Reinforcing miR-215 …

Oct 1, 2021 · Hypoxia restrained the viability, proliferation, migration, and glycolysis and induced the apoptosis, oxidative stress, and inflammation of AC16 cells, and these effects were …

ELISA assay was processed to show the content of IL-6, IL-1ß, IL-10 and TGF-ß in the cell culture supernatant. After being exposed to LPS, cardiomyocyte cells expressed more miR-4763-3p. …

Sep 21, 2021 · To measure the levels of inducible nitric oxide synthase (iNOS), C‐C motif chemokine 2 (CCL2), growth‐regulated alpha protein (CXCL1), and interleukin‐6 (IL‐6) …

May 4, 2024 · AC16 cells were treated with TNF-α, IL-6, and LPS in the presence and absence of CYP1B1-siRNA or resveratrol. mRNA and protein expression levels of CYP1B1 and …

1 day ago · AC16 cells were used to simulate MIRI by glucose-oxygen deprivation, and apoptosis was detected by Annexin V-FITC/PI double staining; protein expression was detected by …

CA promoted the viability and reduced cell apoptosis of AC16 cells and repressed the release of inflammatory cytokines TNF-α, IL-6, and IL-1β, and inhibited the leakage of myocardial injury …

ELISA demonstrated that the amount of TNF-α, IL-1β and IL-6 were enhanced with the increase of LPS concentration, indicating the increased inflammatory response of AC-16 cells ( Fig. 4B-E).

Mar 13, 2025 · A–C qRT-PCR was performed to detect the expression levels of inflammatory factors IL-1β, TNF-α, IL-6, MCP-1 and anti-inflammatory factors IL-10 and TGF-β in each …